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Irbesartan blocks the vasoconstrictor and aldosterone-secreting consequences of angiotensin II by selectively binding to your AT1 angiotensin II receptor. You can find also an AT2 receptor in many tissues, but It isn't involved with cardiovascular homeostasis.
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Irbesartan helps prevent angiotensin II binding to the AT1 receptor in tissues like vascular smooth muscle mass as well as adrenal gland. Irbesartan and its active metabolite bind the AT1 receptor with 8500 times much more affinity than they bind for the AT2 receptor.
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Angiotensin II would or else bind towards the AT1 receptor, inducing vasoconstriction and aldosterone secretion, raising hypertension.
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Because irbesartan will not inhibit ACE, it does not have an impact on the reaction to bradykinin; regardless of whether this has medical relevance is just not recognized.
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